Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. However, our power to rescue SERT functionality in KI midbrain synaptoneurosomes via the inhibition of FAK signifies elevated FAK signaling downstream on the Pro32Pro33 mutant, as verified by increased pFAK localization in 5-HT synapses. Our details https://anthonys764whs5.newsbloger.com/profile