Trans-FA inhibits the proliferation of H1299 lung cancer cells and induces a reasonable increase in the apoptotic inhabitants by promoting phosphorylation of β-catenin at residues Thr41 and Ser45 and producing proteasomal degradation [117,177]. Intriguingly, FA is able to inhibiting the proliferation and migration of lung cancer cells by eliminating intracellular https://trichostatin-a22790.iyublog.com/28878904/astragalin-no-further-a-mystery